par Weiss, Emmanuel;Rautou, Pierre-Emmanuel;Fasseu, Magali;Giabicani, Mikhael;de Chambrun, Marc;Wan, JingHong;Minsart, Charlotte 
;Gustot, Thierry ;Couvineau, Alain;Maiwall, Rakhi;Hurtado-Nedelec, Margarita;Pilard, Nathalie;Lebrec, Didier;Valla, Dominique;Durand, François;de la Grange, Pierre;Monteiro, Renato C;Paugam-Burtz, Catherine;Lotersztajn, Sophie;Moreau, Richard
Référence Journal of hepatology
Publication Publié, 2016-12
;Gustot, Thierry ;Couvineau, Alain;Maiwall, Rakhi;Hurtado-Nedelec, Margarita;Pilard, Nathalie;Lebrec, Didier;Valla, Dominique;Durand, François;de la Grange, Pierre;Monteiro, Renato C;Paugam-Burtz, Catherine;Lotersztajn, Sophie;Moreau, RichardRéférence Journal of hepatology
Publication Publié, 2016-12
Article révisé par les pairs
| Résumé : | In immune cells, constitutively and acutely produced type I interferons (IFNs) engage autocrine/paracrine signaling pathways to induce IFN-stimulated genes (ISGs). Enhanced activity of IFN signaling pathways can cause excessive inflammation and tissue damage. We aimed to investigate the poorly known ISG expression in systemic immune cells from patients with decompensated alcoholic cirrhosis and its association with outcome. |
