Articles dans des revues avec comité de lecture (144)
  1. 81. Sharma, A., Awasthi, S., Harrod, C. K., Matlock, E. F., Khan, S., Xu, L., Chan, S., Yang, H., Thammavaram, C. K., Rasor, R. A., Burns, D. K., Skiest, D. J., Van Lint, C., Girard, A.-M., McGee, M., Monnat, R. J., & Harrod, R. (2007). The Werner syndrome helicase is a cofactor for HIV-1 long terminal repeat transactivation and retroviral replication. The Journal of biological chemistry, 282(16), 12048-12057. doi:10.1074/jbc.M608104200
  2. 82. Horion, J., Gloire, G., El Mjiyad, N., Quivy, V., Vermeulen, L., Vanden Berghe, W., Haegeman, G., Van Lint, C., Piette, J., & Habraken, Y. (2007). Histone deacetylase inhibitor trichostatin A sustains sodium pervanadate-induced NF-kappaB activation by delaying IkappaBalpha mRNA resynthesis: comparison with tumor necrosis factor alpha. The Journal of biological chemistry, 282(21), 15383-15393. doi:10.1074/jbc.M609166200
  3. 83. Marban, C., Suzanne, S., Dequiedt, F., De Walque, S., Redel, L., Van Lint, C., Aunis, D., & Rohr, O. (2007). Recruitment of chromatin-modifying enzymes by CTIP2 promotes HIV-1 transcriptional silencing. EMBO journal, 26(2), 412-423. doi:10.1038/sj.emboj.7601516
  4. 84. Vandergeeten, C., Quivy, V., Moutschen, M., Van Lint, C., Piette, J., & Legrand-Poels, S. (2007). HIV-1 protease inhibitors do not interfere with provirus transcription and host cell apoptosis induced by combined treatment TNF-alpha + TSA. Biochemical pharmacology, 73(11), 1738-1748. doi:10.1016/j.bcp.2007.02.011
  5. 85. Nguyen, T. L.-A., De Walque, S., Veithen, E., Dekoninck, A., Martinelli, V., De Launoit, Y., Burny, A., Harrod, R., & Van Lint, C. (2007). Transcriptional regulation of the bovine leukemia virus promoter by the cyclic AMP-response element modulator tau isoform. The Journal of biological chemistry, 282(29), 20854-20867. doi:10.1074/jbc.M703060200
  6. 86. Quivy, V., De Walque, S., & Van Lint, C. (2007). Chromatin-associated regulation of HIV-1 transcription: implications for the development of therapeutic strategies. Sub-cellular biochemistry, 41, 371-396.
  7. 87. Castellano, R., Vire, B., Pion, M., Quivy, V., Olive, D., Hirsch, I., Van Lint, C., & Collette, Y. (2006). Active transcription of the human FASL/CD95L/TNFSF6 promoter region in T lymphocytes involves chromatin remodeling: role of DNA methylation and protein acetylation suggest distinct mechanisms of transcriptional repression. The Journal of biological chemistry, 281(21), 14719-14728. doi:10.1074/jbc.M602373200
  8. 88. Mauen, S., Huvent, I., Raussens, V., Demonte, D., Baert, J.-L., Tricot, C., Ruysschaert, J. M., Van Lint, C., Moguilevsky, N., & De Launoit, Y. (2006). Expression, purification, and structural prediction of the Ets transcription factor ERM. Biochimica et biophysica acta, 1760(8), 1192-1201. doi:10.1016/j.bbagen.2006.03.007
  9. 89. Wong, K., Sharma, A., Awasthi, S., Matlock, E. F., Rogers, L., Van Lint, C., Skiest, D. J., Burns, D. K., & Harrod, R. (2005). HIV-1 Tat interactions with p300 and PCAF transcriptional coactivators inhibit histone acetylation and neurotrophin signaling through CREB. The Journal of biological chemistry, 280(10), 9390-9399. doi:10.1074/jbc.M408643200
  10. 90. T'Sas, F., Brenner, C., Mauen, S., Putmans, P., Monte, D., Van Lint, C., Moser, M., Baert, J.-L., & De Launoit, Y. (2005). Expression of the Ets transcription factor Erm is regulated through a conventional PKC signaling pathway in the Molt4 lymphoblastic cell line. FEBS letters, 579(1), 66-70. doi:10.1016/j.febslet.2004.11.052
  11. 91. Marban, C., Redel, L., Suzanne, S., Van Lint, C., Lecestre, D., Chasserot-Golaz, S., Leid, M., Aunis, D., Schaeffer, E., & Rohr, O. (2005). COUP-TF interacting protein 2 represses the initial phase of HIV-1 gene transcription in human microglial cells. Nucleic acids research, 33(7), 2318-2331. doi:10.1093/nar/gki529
  12. 92. Kim, Y.-S., Kim, J.-M., Jung, D.-L., Kang, J.-E., Lee, S., Kim, J. S., Seol, W., Shin, H.-C., Kwon, H. S., Van Lint, C., Hernandez, N., & Hur, M.-W. (2005). Artificial zinc finger fusions targeting Sp1-binding sites and the trans-activator-responsive element potently repress transcription and replication of HIV-1. The Journal of biological chemistry, 280(22), 21545-21552. doi:10.1074/jbc.M414136200
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