Articles dans des revues avec comité de lecture (144)
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Bellefroid, M., Rodari, A., Galais, M., Krijger, P., Tjalsma, S., Nestola, L., Plant, E., Vos, E., Cristinelli, S. S., Van Driessche, B., Vanhulle, C., Ait-Ammar, A., Burny, A., Ciuffi, A., De Laat, W., & Van Lint, C. (2022). Role of the cellular factor CTCF in the regulation of bovine leukemia virus latency and three-dimensional chromatin organization. Nucleic acids research, 50(6), 3190-3202. doi:10.1093/nar/gkac10712.
Hamann, M. M., Ehmele, P., Verdikt, R., Bialek-Waldmann, J. J., Virdi, S., Günther, T., Van Lint, C., Grundhoff, A., Hauber, J., & Lange, U. C. (2021). Transcriptional behavior of the HIV-1 promoter in context of the BACH2 prominent proviral integration gene. Virus research, 293, 198260. doi:10.1016/j.virusres.2020.19826015.
Ait-Ammar, A., Bellefroid, M., Daouad, F., Martinelli, V., Van Assche, J., Wallet, C., Rodari, A., De Rovere, M., Fahrenkrog, B., Schwartz, C., Van Lint, C., Gautier, V., & Rohr, O. (2021). Inhibition of HIV-1 gene transcription by KAP1 in myeloid lineage (C. Van Lint, V. Gautier and O. Rohr are co-last authors). Scientific reports, 11(1), 2692. doi:10.1038/s41598-021-82164-w17.
Ait-Ammar, A., Kula, A., Darcis, G., Verdikt, R., De Wit, S., Gautier, V., Mallon, P., Marcello, A., Rohr, O., & Van Lint, C. (2020). Current Status of Latency Reversing Agents Facing the Heterogeneity of HIV-1 Cellular and Tissue Reservoirs. Frontiers in microbiology, 10, 3060. doi:10.3389/fmicb.2019.0306019.
Darcis, G., Kootstra, N., Hooibrink, B., Van Montfort, T., Maurer, I., Groen, K., Jurriaans, S., Bakker, M., Van Lint, C., Berkhout, B., & Pasternak, A. (2020). CD32+CD4+ T Cells Are Highly Enriched for HIV DNA and Can Support Transcriptional Latency. Cell reports, 30(7), 2284-2296.e3. doi:10.1016/j.celrep.2020.01.07120.
Forouzanfar, F., Ali, S., Wallet, C., De Rovere, M., Ducloy, C., El Mekdad, H., El Maassarani, M., Aït-Ammar, A., Van Assche, J., Boutant, E., Daouad, F., Margottin-Goguet, F., Moog, C., Van Lint, C., Schwartz, C., & Rohr, O. (2019). HIV-1 Vpr mediates the depletion of the cellular repressor CTIP2 to counteract viral gene silencing. Scientific reports, 9(1), 13154. doi:10.1038/s41598-019-48689-x