Articles dans des revues avec comité de lecture (144)
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Critchfield, J. W., Ho, O., Roberts, M., Van Lint, C., Verdin, E., & Butera, S. T. (1999). Isoquinolinesulphonamide derivatives inhibit transcriptional elongation of human immunodeficiency virus type 1 RNA in a promyelocytic model of latency. Antiviral chemistry & chemotherapy, 10(5), 275-284.130.
Chariot, A., Van Lint, C., Chapelier, M., Gielen, J., Merville, M.-P., & Bours, V. (1999). CBP and histone deacetylase inhibition enhance the transactivation potential of the HOXB7 homeodomain-containing protein. Oncogene, 18(27), 4007-4014. doi:10.1038/sj.onc.1202776131.
Kiernan, R. E., Vanhulle, C., Schiltz, L., Adam, E., Xiao, H., Maudoux, F., Calomme, C., Burny, A., Nakatani, Y., Jeang, K. T., Benkirane, M. M., & Van Lint, C. (1999). HIV-1 tat transcriptional activity is regulated by acetylation. EMBO journal, 18(21), 6106-6118. doi:10.1093/emboj/18.21.6106132.
Emiliani, S., Fischle, W., Van Lint, C., Al-Abed, Y., & Verdin, E. (1998). Characterization of a human RPD3 ortholog, HDAC3. Proceedings of the National Academy of Sciences of the United States of America, 95(6), 2795-2800.133.
Kiermer, V., Van Lint, C., Briclet, D., Vanhulle, C., Kettmann, R., Verdin, E., Burny, A., & Droogmans, L. (1998). An interferon regulatory factor binding site in the U5 region of the bovine leukemia virus long terminal repeat stimulates Tax-independent gene expression. Journal of virology, 72(7), 5526-5534.135.
Emiliani, S., Fischle, W., Ott, M., Van Lint, C., Amella, C. A., & Verdin, E. (1998). Mutations in the tat gene are responsible for human immunodeficiency virus type 1 postintegration latency in the U1 cell line. Journal of virology, 72(2), 1666-1670.136.
Ott, M., Emiliani, S., Van Lint, C., Herbein, G., Lovett, J., Chirmule, N., McCloskey, T., Pahwa, S., & Verdin, E. (1997). Immune hyperactivation of HIV-1-infected T cells mediated by Tat and the CD28 pathway. Science, 275(5305), 1481-1485.137.
Van Lint, C., Amella, C. A., Emiliani, S., John, M., Jie, T., & Verdin, E. (1997). Transcription factor binding sites downstream of the human immunodeficiency virus type 1 transcription start site are important for virus infectivity. Journal of virology, 71(8), 6113-6127.140.
Emiliani, S., Van Lint, C., Fischle, W., Paras, P., Ott, M., Brady, J., & Verdin, E. (1996). A point mutation in the HIV-1 Tat responsive element is associated with postintegration latency. Proceedings of the National Academy of Sciences of the United States of America, 93(13), 6377-6381.