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Diesel exhaust exposure favors TH2 cell recruitment in nonatopic subjects by differentially regulating chemokine production.

par Chang, Ying;Sénéchal, Stéphanie;De Nadai, Patricia ;Chenivesse, Cécile;Gilet, Jules;Vorng, Han;Legendre, Benjamin;Tonnel, André-Bernard;Wallaert, Benoît;Lassalle, Philippe;Tsicopoulos, Anne
Référence Journal of allergy and clinical immunology, 118, 2, page (354-360)
Publication Publié, 2006-08

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Titre:	Diesel exhaust exposure favors TH2 cell recruitment in nonatopic subjects by differentially regulating chemokine production.
Auteur:	Chang, Ying; Sénéchal, Stéphanie; De Nadai, Patricia; Chenivesse, Cécile; Gilet, Jules; Vorng, Han; Legendre, Benjamin; Tonnel, André-Bernard; Wallaert, Benoît; Lassalle, Philippe; Tsicopoulos, Anne
Informations sur la publication:	Journal of allergy and clinical immunology, 118, 2, page (354-360)
Statut de publication:	Publié, 2006-08
Sujet CREF:	Sciences bio-médicales et agricoles
MeSH keywords:	Adult
	Cells, Cultured
	Chemokines -- biosynthesis
	Chemotaxis, Leukocyte -- drug effects
	Humans
	Hypersensitivity -- etiology -- immunology
	Leukocytes, Mononuclear -- drug effects -- metabolism
	Middle Aged
	Polycyclic Hydrocarbons, Aromatic -- toxicity
	Th1 Cells -- drug effects -- immunology
	Th2 Cells -- drug effects -- immunology
	Vehicle Emissions -- toxicity
Note générale:	Journal Article
	Research Support, Non-U.S. Gov't
Langue:	Anglais
Identificateurs:	urn:issn:0091-6749
	info:doi/10.1016/j.jaci.2006.04.050
	info:pii/S0091-6749(06)00945-6
	info:pmid/16890758