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Extracellular signal-regulated kinase is essential for interleukin-1-induced and nuclear factor kappaB-mediated gene expression in insulin-producing INS-1E cells.

par Larsen, L;Størling, Joachim;Darville, Martine ;Eizirik, Decio L. ;Bonny, Christophe;Billestrup, N;Mandrup-Poulsen, T.
Référence Diabetologia, 48, 12, page (2582-2590)
Publication Publié, 2005

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Titre:	Extracellular signal-regulated kinase is essential for interleukin-1-induced and nuclear factor kappaB-mediated gene expression in insulin-producing INS-1E cells.
Auteur:	Larsen, L; Størling, Joachim; Darville, Martine; Eizirik, Decio L.; Bonny, Christophe; Billestrup, N; Mandrup-Poulsen, T.
Informations sur la publication:	Diabetologia, 48, 12, page (2582-2590)
Statut de publication:	Publié, 2005
Sujet CREF:	Sciences bio-médicales et agricoles
Mots-clés:	IL-1β
	Mitogen-activated protein kinases
	Nitric oxide
	Nuclear factor kappa B
	Pancreatic beta cells
	Type 1 diabetes
MeSH keywords:	Animals
	Apoptosis -- genetics
	Blotting, Western
	Cell Line, Tumor
	Extracellular Signal-Regulated MAP Kinases -- antagonists & inhibitors
	Extracellular Signal-Regulated MAP Kinases -- physiology
	Flavonoids -- pharmacology
	Gene Expression -- drug effects
	Gene Expression -- physiology
	Humans
	Imidazoles -- pharmacology
	Insulin -- secretion
	Insulin-Secreting Cells -- drug effects
	Insulin-Secreting Cells -- physiology
	Insulin-Secreting Cells -- secretion
	Interleukin-1 -- pharmacology
	Interleukin-1 -- physiology
	MAP Kinase Kinase 4 -- antagonists & inhibitors
	MAP Kinase Kinase 4 -- physiology
	Mitogen-Activated Protein Kinase Kinases -- antagonists & inhibitors
	Mitogen-Activated Protein Kinase Kinases -- physiology
	NF-kappa B -- physiology
	Nitric Oxide Synthase Type II -- biosynthesis
	Nitric Oxide Synthase Type II -- genetics
	Phosphorylation
	Pyridines -- pharmacology
	Rats
	Serine -- analysis
	Synaptotagmin I -- chemistry
	Synaptotagmin I -- physiology
	p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
	p38 Mitogen-Activated Protein Kinases -- physiology
Note générale:	Journal Article
	Research Support, Non-U.S. Gov't
Langue:	Anglais
Identificateurs:	urn:issn:0012-186X
	info:doi/10.1007/s00125-005-0039-9
	info:scp/28444472055
	info:pmid/16283237