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Interleukin-1beta-induced alteration in a beta-cell phenotype can reduce cellular sensitivity to conditions that cause necrosis but not to cytokine-induced apoptosis.

par Ling, Z;Van De Casteele, Christine ;Eizirik, Decio L. ;Pipeleers, Daniel
Référence Diabetes (New York, N.Y.), 49, 3, page (340-345)
Publication Publié, 2000

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Titre:	Interleukin-1beta-induced alteration in a beta-cell phenotype can reduce cellular sensitivity to conditions that cause necrosis but not to cytokine-induced apoptosis.
Auteur:	Ling, Z; Van De Casteele, Christine; Eizirik, Decio L.; Pipeleers, Daniel
Informations sur la publication:	Diabetes (New York, N.Y.), 49, 3, page (340-345)
Statut de publication:	Publié, 2000
Sujet CREF:	Sciences bio-médicales et agricoles
MeSH keywords:	Alloxan -- pharmacology
	Animals
	Apoptosis -- physiology
	Cytokines -- physiology
	Dose-Response Relationship, Drug
	Interleukin-1 -- pharmacology
	Islets of Langerhans -- drug effects
	Islets of Langerhans -- pathology
	Islets of Langerhans -- physiology
	Male
	Necrosis
	Nitric Oxide Donors -- pharmacology
	Osmolar Concentration
	Phenotype
	Proteins -- metabolism
	Rats
	Rats, Wistar
	Streptozocin -- pharmacology
	Triazoles -- pharmacology
Note générale:	Journal Article
	Research Support, Non-U.S. Gov't
Langue:	Anglais
Identificateurs:	urn:issn:0012-1797
	info:pmid/10868954