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S100A6 overexpression within astrocytes associated with impaired axons from both ALS mouse model and human patients.

par Hoyaux, Daphné ;Boom, Alain ;Van Den Bosch, L;Belot, Nathalie ;Martin, Jean-Jacques ;Heizmann, Claus W;Kiss, Robert ;Pochet, Roland
Référence Journal of neuropathology and experimental neurology, 61, 8, page (736-744)
Publication Publié, 2002

Article révisé par les pairs

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Titre:	S100A6 overexpression within astrocytes associated with impaired axons from both ALS mouse model and human patients.
Auteur:	Hoyaux, Daphné; Boom, Alain; Van Den Bosch, L; Belot, Nathalie; Martin, Jean-Jacques; Heizmann, Claus W; Kiss, Robert; Pochet, Roland
Informations sur la publication:	Journal of neuropathology and experimental neurology, 61, 8, page (736-744)
Statut de publication:	Publié, 2002
Sujet CREF:	Sciences bio-médicales et agricoles
Mots-clés:	Amyotrophic lateral sclerosis
	Astrocytes
	Calcium binding proteins
	Glial fibrillary associated protein (GFAP)
	S100A6
	SOD1
	Zn2+
MeSH keywords:	Adult
	Aged
	Amyotrophic Lateral Sclerosis -- genetics
	Amyotrophic Lateral Sclerosis -- metabolism
	Amyotrophic Lateral Sclerosis -- pathology
	Animals
	Astrocytes -- metabolism
	Axons -- pathology
	Cell Cycle Proteins
	Central Nervous System -- metabolism
	Central Nervous System -- pathology
	Disease Models, Animal
	Female
	Gene Expression
	Glial Fibrillary Acidic Protein -- metabolism
	Humans
	Male
	Mice
	Mice, Transgenic -- genetics
	Middle Aged
	Motor Neurons -- pathology
	Mutation
	S100 Proteins -- metabolism
	Superoxide Dismutase -- genetics
Note générale:	Journal Article
	Research Support, Non-U.S. Gov't
Langue:	Anglais
Identificateurs:	urn:issn:0022-3069
	info:scp/0036328444
	info:pmid/12152788