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Insulin increase in MAP kinase phosphorylation is shifted to early time-points by overexpressing APS, while Akt phosphorylation is not influenced.

par Onnockx, SHEELA ;Xie, Jingwei ;Degraef, Chantal ;Erneux, Christophe ;Pirson, Isabelle
Référence Experimental cell research, 315, 15, page (2479-2486)
Publication Publié, 2009-09

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Titre:	Insulin increase in MAP kinase phosphorylation is shifted to early time-points by overexpressing APS, while Akt phosphorylation is not influenced.
Auteur:	Onnockx, SHEELA; Xie, Jingwei; Degraef, Chantal; Erneux, Christophe; Pirson, Isabelle
Informations sur la publication:	Experimental cell research, 315, 15, page (2479-2486)
Statut de publication:	Publié, 2009-09
Sujet CREF:	Sciences bio-médicales et agricoles
Mots-clés:	Adaptor protein APS
	Insulin
	MAPK
	PI 3-kinase
	PKB
	SHIP2
MeSH keywords:	1-Phosphatidylinositol 3-Kinase -- antagonists & inhibitors
	1-Phosphatidylinositol 3-Kinase -- genetics
	1-Phosphatidylinositol 3-Kinase -- metabolism
	Adaptor Proteins, Signal Transducing -- genetics
	Adaptor Proteins, Signal Transducing -- metabolism
	Animals
	CHO Cells
	Cell Membrane -- metabolism
	Cricetinae
	Cricetulus
	Enzyme Activation
	Inositol Phosphates -- metabolism
	Insulin -- metabolism
	Mitogen-Activated Protein Kinases -- metabolism
	Mutagenesis, Site-Directed
	Phosphorylation
	Point Mutation
	Proto-Oncogene Proteins c-akt -- metabolism
	Signal Transduction -- physiology
	src Homology Domains
Note générale:	Journal Article
	Research Support, Non-U.S. Gov't
Langue:	Anglais
Identificateurs:	urn:issn:0014-4827
	info:doi/10.1016/j.yexcr.2009.06.006
	info:pii/S0014-4827(09)00269-9
	info:scp/67651062293
	info:pmid/19527711