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Deletion of STAT-1 pancreatic islets protects against streptozotocin-induced diabetes and early graft failure but not against late rejection.

par Callewaert, Hanne;Gysemans, Conny A;Ladrière, Laurence ;D'Hertog, Wannes;Hagenbrock, Julianna;Overbergh, Lutgart;Eizirik, Decio L. ;Mathieu, Chantal
Référence Diabetes (New York, N.Y.), 56, 8, page (2169-2173)
Publication Publié, 2007

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Titre:	Deletion of STAT-1 pancreatic islets protects against streptozotocin-induced diabetes and early graft failure but not against late rejection.
Auteur:	Callewaert, Hanne; Gysemans, Conny A; Ladrière, Laurence; D'Hertog, Wannes; Hagenbrock, Julianna; Overbergh, Lutgart; Eizirik, Decio L.; Mathieu, Chantal
Informations sur la publication:	Diabetes (New York, N.Y.), 56, 8, page (2169-2173)
Statut de publication:	Publié, 2007
Sujet CREF:	Sciences bio-médicales et agricoles
MeSH keywords:	Alloxan -- pharmacology
	Animals
	Diabetes Mellitus, Experimental -- chemically induced
	Diabetes Mellitus, Experimental -- immunology
	Diabetes Mellitus, Experimental -- metabolism
	Diabetes Mellitus, Experimental -- pathology
	Dose-Response Relationship, Drug
	Gene Expression Regulation
	Graft Rejection -- immunology
	Graft Rejection -- prevention & control
	Interleukin-1beta -- metabolism
	Islets of Langerhans Transplantation -- immunology
	Mice
	Mice, Knockout
	STAT1 Transcription Factor -- deficiency
	STAT1 Transcription Factor -- genetics
	STAT1 Transcription Factor -- metabolism
	Signal Transduction -- drug effects
	Streptozocin -- pharmacology
	Survival Rate
	Time Factors
Note générale:	Journal Article
	Research Support, Non-U.S. Gov't
Langue:	Anglais
Identificateurs:	urn:issn:0012-1797
	info:doi/10.2337/db07-0052
	info:pii/db07-0052
	info:scp/34547561721
	info:pmid/17473223