par Haot, Jules 
;Jouret, Anne
Référence Acta endoscopica, 32, 2, page (125-132)
Publication Publié, 2002
;Jouret, AnneRéférence Acta endoscopica, 32, 2, page (125-132)
Publication Publié, 2002
Article révisé par les pairs
| Résumé : | Helicobacter pylori represents the overwhelming part of gastric infectious pathology leaving far behind all the other bacteria. After a short acute phase, most Hp gastritis evolve on a chronic mode. Their main histological characteristics are made of epithelial degenerative and regenerative alterations and cellular inflammatory response of the lamina propria. The lesions can be graded according to the Sydney System (revisited Houston from 1994). The detection of Hp can rely upon special stains such as Giemsa or Cresyl Violet. Immunocytochemistry can be helpful when the germs are scarce (some acute gastritis, after antibiotic treatment…). Another much less frequent Helicobacter is Helicobacter heilmannii which probably results from contaminations by pets and produces lesions very similar to those of Hp. CMV can be observed in immunocompetent as well as immunodeficient hosts; it induces a variety of lesions, sometimes mimicking cancer. Various fungi can localise to the stomach mostly in necrotic tissues from cancers or ulcers. In immunodeficient patients, the lesions can extend to various systems. The other germs, sometimes observed in the stomach, either bacterial, fungal or parasitic are quite rare. |
