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Connexin36 contributes to INS-1E cells survival through modulation of cytokine-induced oxidative stress, ER stress and AMPK activity.

par Allagnat, Florent;Klee, Philippe;Cardozo, Alessandra K ;Meda, P;Haefliger, J A
Référence Cell death and differentiation, 20, 12, page (1742-1752)
Publication Publié, 2013-12

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Titre:	Connexin36 contributes to INS-1E cells survival through modulation of cytokine-induced oxidative stress, ER stress and AMPK activity.
Auteur:	Allagnat, Florent; Klee, Philippe; Cardozo, Alessandra K; Meda, P; Haefliger, J A
Informations sur la publication:	Cell death and differentiation, 20, 12, page (1742-1752)
Statut de publication:	Publié, 2013-12
Sujet CREF:	Sciences bio-médicales et agricoles
MeSH keywords:	AMP-Activated Protein Kinases -- metabolism
	Animals
	Apoptosis -- drug effects
	Calcium -- metabolism
	Caspase 3 -- metabolism
	Cell Survival -- drug effects
	Connexins -- genetics -- metabolism
	Cyclic AMP -- metabolism
	Cytokines -- pharmacology
	Down-Regulation -- drug effects
	Endoplasmic Reticulum Stress -- drug effects
	Feedback, Physiological -- drug effects
	Humans
	Insulin -- biosynthesis
	Insulin-Secreting Cells -- drug effects -- metabolism -- pathology
	Interleukin-1beta -- pharmacology
	Metformin -- pharmacology
	Mice
	Mitochondria -- drug effects -- metabolism
	Models, Biological
	Oxidative Stress -- drug effects
	Rats
	Reactive Nitrogen Species -- metabolism
	Reactive Oxygen Species -- metabolism
	Response Elements -- genetics
	Transcription, Genetic -- drug effects
Note générale:	Journal Article
	Research Support, N.I.H., Extramural
	Research Support, Non-U.S. Gov't
	SCOPUS: ar.j
Langue:	Anglais
Identificateurs:	urn:issn:1350-9047
	info:doi/10.1038/cdd.2013.134
	info:pii/cdd2013134
	info:scp/84887620071
	info:pmid/24096873