Articles dans des revues avec comité de lecture (405)
  1. 356. Dunger, A., Sjöholm, A., & Eizirik, D. L. (1990). Amino acids and human amniotic fluid increase DNA biosynthesis in pancreatic islets of adult mouse, but this effect is lost following exposure to streptozotocin. Pancreas, 5(6), 639-646.
  2. 357. Strandell, E., Eizirik, D. L., & Sandler, S. (1990). Reversal of beta-cell suppression in vitro in pancreatic islets isolated from nonobese diabetic mice during the phase preceding insulin-dependent diabetes mellitus. The Journal of clinical investigation, 85(6), 1944-1950. doi:10.1172/JCI114657
  3. 358. Eizirik, D. L., Welsh, M., Strandell, E., Welsh, N., & Sandler, S. (1990). Interleukin-1 beta depletes insulin messenger ribonucleic acid and increases the heat shock protein hsp70 in mouse pancreatic islets without impairing the glucose metabolism. Endocrinology, 127(5), 2290-2297.
  4. 359. Borg, L. A., & Eizirik, D. L. (1990). Short-term exposure of rat pancreatic islets to human interleukin-1 beta increases cellular uptake of calcium. Immunology letters, 26(3), 253-258.
  5. 360. Sandler, S., Bendtzen, K., Eizirik, D. L., Strandell, E., Welsh, M., & Welsh, N. (1990). Metabolism and beta-cell function of rat pancreatic islets exposed to human interleukin-1 beta in the presence of a high glucose concentration. Immunology letters, 26(3), 245-251.
  6. 361. Jansson, L., Eizirik, D. L., & Sandler, S. (1989). Terbutaline decreases the blood flow of the pancreatic islets but does not reduce the diabetogenic action of streptozotocin in the rat. European journal of pharmacology, 161(1), 79-83.
  7. 362. Strandell, E., Eizirik, D. L., & Sandler, S. (1989). Survival and B-cell function of mouse pancreatic islets maintained in culture after concomitant exposure to streptozotocin and nicotinamide. Experimental and clinical endocrinology, 93(2-3), 219-224. doi:10.1055/s-0029-1210860
  8. 363. Sandler, S., Bendtzen, K., Borg, L. A., Eizirik, D. L., Strandell, E., & Welsh, N. (1989). Studies on the mechanisms causing inhibition of insulin secretion in rat pancreatic islets exposed to human interleukin-1 beta indicate a perturbation in the mitochondrial function. Endocrinology, 124(3), 1492-1501.
  9. 364. Eizirik, D. L., & Sandler, S. (1989). Sustained exposure of toxically damaged mouse pancreatic islets to high glucose does not increase beta-cell dysfunction. Journal of Endocrinology, 123(1), 47-51.
  10. 365. Eizirik, D. L., Sandler, S., Korsgren, O., Jansson, L., & Andersson, A. (1989). Persistent impairment of the insulin response to glucose both in vivo and in vitro after streptozotocin exposure: studies with grafted pancreatic islets and islets maintained in culture. Acta endocrinologica (Print ed.), 121(6), 849-856.
  11. 366. Eizirik, D. L., & Sandler, S. (1989). Function and metabolism of pancreatic beta-cells maintained in culture following experimentally induced damage. Pharmacology & toxicology, 65(3), 163-168.
  12. 367. Eizirik, D. L., Sandler, S., Hallberg, A., Bendtzen, K., Sener, A., & Malaisse, W. (1989). Differential sensitivity to beta-cell secretagogues in cultured rat pancreatic islets exposed to human interleukin-1 beta. Endocrinology, 125(2), 752-759. doi:10.1210/endo-125-2-752
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