Parties d'ouvrages collectifs (5)

  1. 1. Eizirik, D. L., & Hoorens, A. (1999). Beta-cell dysfunction and death. In S. L. Howell (Ed.), Biology of the pancreatic beta-cell (pp. 47-73). Connecticut, USA: JAI Press INC.(Advances in molecular and cell biology, 29).
  2. 2. Sandler, S., & Eizirik, D. L. (1996). Culture of human pancreatic islet cells. In G. D. Jones (Ed.), Human cell culture protocols (pp. 391-4017). Totowa, USA: Humana Press.(Methods in Molecular Medicine, 2).
  3. 3. Eizirik, D. L., Sandler, S., Björklund, A., & Welsh, N. (1994). Possible mechanisms of damage and repair of pancreatic beta-cells in diabetes mellitus. In P. Flatt & S. Lenzen (Eds.), Frontiers of pancreatic beta-cell research (pp. 535-542). London: Smith Gordon and Co. Ldt.
  4. 4. Sandler, S., Eizirik, D. L., Sternesjö, J., & Welsh, N. (1994). Cytokines and islet beta-cell function. In P. Flatt & S. Lenzen (Eds.), Frontiers of pancreatic beta-cell research (pp. 553-558). London: Smith Gordon and Co. Ldt.
  5. 5. Eizirik, D. L., & Hellerström, C. (1993). Structure and function of the normal and diabetic beta-cell. In A. K. Sharma (Ed.), Diabetes mellitus and its complications: an update (pp. 40-50). London: Macmillan International.
  6.   Articles dans des revues avec comité de lecture (400)

  7. 1. Eizirik, D. L., Szymczak, F., & Mallone, R. (2023). Why does the immune system destroy pancreatic β- but not α-cells in type 1 diabetes? Nature reviews. Endocrinology.
  8. 2. Zhang, J., Zou, Y., Chen, L., Sun, F., Xu, Q., Zhou, Q., Wang, Y., Luo, X., Wang, N., Li, Y., Zhang, S., Xiong, F., Yang, P., Liu, S.-W., Yang, T., Weng, J., Eizirik, D. L., Yan, J., Zhou, Z., & Wang, C. Y. (2023). Myo9b mutations are associated with altered dendritic cell functions and increased susceptibility to autoimmune diabetes onset. Nature communications, 14(1), 5977. doi:10.1038/s41467-023-41534-w
  9. 3. Cantley, J., Eizirik, D. L., Latres, E., & Dayan, C. C. (2023). Islet cells in human type 1 diabetes: from recent advances to novel therapies - a symposium-based roadmap for future research. The Journal of endocrinology, 259(1). doi:10.1530/JOE-23-0082
  10. 4. Castellá, M., Blasco-Roset, A., Peyrou, M., Gavaldà-Navarro, A., Villarroya, J., Quesada-López, T., Lorente-Poch, L., Sancho, J. M., Szymczak, F., Piron, A., Rodríguez-Fernández, S., Carobbio, S., Goday, A., Domingo, P., Vidal-Puig, A., Giralt, M., Eizirik, D. L., Villarroya, F., & Cereijo, R. (2023). Adipose tissue plasticity in pheochromocytoma patients suggests a role of the splicing machinery in human adipose browning. iScience, 26(6), 106847. doi:10.1016/j.isci.2023.106847
  11. 5. Griess, K., Rieck, M., Müller, N., Karsai, G., Hartwig, S., Pelligra, A., Hardt, R., Schlegel, C., Kuboth, J., Uhlemeyer, C., Trenkamp, S., Jeruschke, K., Weiss, J., Peifer-Weiss, L., Xu, W., Cames, S., Yi, X., Cnop, M., Beller, M., Stark, H., Kondadi, A. K., Reichert, A., Markgraf, D., Wammers, M., Häussinger, D., Kuss, O., Lehr, S., Eizirik, D. L., Lickert, H., Lammert, E., Roden, M., Winter, D., Al-Hasani, H., Höglinger, D., Hornemann, T., Brüning, J., & Belgardt, B.-F. (2023). Sphingolipid subtypes differentially control proinsulin processing and systemic glucose homeostasis. Nature cell biology, 25(1), 20-29. doi:10.1038/s41556-022-01027-2
  12. 6. Chandra, V., Ibrahim, H., Halliez, C., Prasad, R., Vecchio, F., Dwivedi, O. P., Kvist, J., Balboa, D., Saarimäki-Vire, J., Montaser, H. E.-D., Barsby, T., Lithovius, V., Artner, I., Gopalakrishnan, S., Groop, L., Mallone, R., Eizirik, D. L., & Otonkoski, T. (2022). The type 1 diabetes gene TYK2 regulates β-cell development and its responses to interferon-α. Nature communications, 13(1), 6363. doi:10.1038/s41467-022-34069-z
  13. 7. Szymczak, F., Cohen-Fultheim, R., Thomaidou, S., Coomans De Brachene, A., Oliveira Castela, A. S., Colli, M. L., Marchetti, P., Levanon, E., Eizirik, D. L., & Zaldumbide, A. A. (2022). ADAR1-dependent editing regulates human β cell transcriptome diversity during inflammation. Frontiers in endocrinology, 13. doi:10.3389/fendo.2022.1058345

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