• Citer

Radiation response of chemically derived mitochondrial DNA-deficient AG01522 human primary fibroblasts.

par Nieri, D;Fioramonti, Marco ;Berardinelli, F;Léonet, Stéphanie;Cherubini, R;De Nadal, V;Gerardi, S;Moreno, S;Nardacci, R;Tanzarella, C;Antoccia, A
Référence Mutation research, 756, 1-2, page (86-94)
Publication Publié, 2013-08

Article révisé par les pairs

• Accès en ligne
• Détails
• Contenu
• Statistiques

Titre:	Radiation response of chemically derived mitochondrial DNA-deficient AG01522 human primary fibroblasts.
Auteur:	Nieri, D; Fioramonti, Marco; Berardinelli, F; Léonet, Stéphanie; Cherubini, R; De Nadal, V; Gerardi, S; Moreno, S; Nardacci, R; Tanzarella, C; Antoccia, A
Informations sur la publication:	Mutation research, 756, 1-2, page (86-94)
Statut de publication:	Publié, 2013-08
Sujet CREF:	Sciences bio-médicales et agricoles
Mots-clés:	Cytogenetic damage
	Human primary fibroblasts
	Ionizing radiations
	Mitochondria
	Mitochondrial fragmentation
	ROS
MeSH keywords:	Antimetabolites -- pharmacology
	Cell Survival -- radiation effects
	Cells, Cultured
	Chromosome Aberrations
	DNA Damage -- genetics -- radiation effects
	DNA Repair -- genetics -- radiation effects
	DNA, Mitochondrial -- drug effects -- metabolism -- radiation effects
	Fibroblasts -- cytology -- metabolism -- radiation effects
	Humans
	In Situ Hybridization, Fluorescence
	Linear Energy Transfer
	Membrane Potential, Mitochondrial -- radiation effects
	Mitochondria -- drug effects -- genetics -- radiation effects
	Reactive Oxygen Species -- metabolism
	Telomere -- genetics
	X-Rays
	Zalcitabine -- pharmacology
Langue:	Anglais
Identificateurs:	urn:issn:0027-5107
	info:doi/10.1016/j.mrgentox.2013.05.010
	info:pii/S1383-5718(13)00130-7
	info:pmid/23721903