Articles dans des revues avec comité de lecture (183)

  1. 26. Gankam Kengne, F., Couturier, B., Soupart, A., Brion, J. P., & Decaux, G. (2017). Osmotic Stress-Induced Defective Glial Proteostasis Contributes to Brain Demyelination after Hyponatremia Treatment. Journal of the American Society of Nephrology, 28(6), 1802-1813. doi:10.1681/ASN.2016050509
  2. 27. Nouini, A., Gaspard, N., Fery, P., Slama, H., Kavec, M., Goldman, S., Brion, J. P., & Bier, J. C. (2017). Semantic Dementia and Motor Neuron Disease: Case Confirmation of TDP43 Pathology Associated with a Predominant Right Temporal Atrophy. EC Neurology, 6(3), 146-152.
  3. 28. Vanden Dries, V., Stygelbout, V., Pierrot, N., Yilmaz, Z., Suain, V., De Decker, R., Buée, L., Octave, J. N., Brion, J. P., & Leroy, K. (2017). Amyloid precursor protein reduction enhances the formation of neurofibrillary tangles in a mutant tau transgenic mouse model. Neurobiology of aging. doi:10.1016/j.neurobiolaging.2017.03.031
  4. 29. Doshina, A., Gourgue, F., Onizuka, M., Opsomer, R., Wang, J.-P., Ando, K., Tasiaux, B., Dewachter, I., Kienlen-Campard, P. K., Brion, J. P., Gailly, P., Octave, J. N., & Pierrot, N. (2017). Cortical cells reveal APP as a new player in the regulation of GABAergic neurotransmission. Scientific reports, 7(1), 370. doi:10.1038/s41598-017-00325-2
  5. 30. Espuny Camacho, I. M., Arranz, A. M., Fiers, M., Snellinx, A., Ando, K., Munck, S., Bonnefont, J., Lambot, L., Corthout, N., Omodho, L., Vanden Eynden, E., Radaelli, E., Tesseur, I., Wray, S., Ebneth, A., Hardy, J., Leroy, K., Brion, J. P., Vanderhaeghen, P., & De Strooper, B. (2017). Hallmarks of Alzheimer's Disease in Stem-Cell-Derived Human Neurons Transplanted into Mouse Brain. Neuron, 93(5), 1066-1081.e8. doi:10.1016/j.neuron.2017.02.001
  6. 31. Poncelet, L., Garigliany, M., Ando, K., Franssen, M., Desmecht, D., & Brion, J. P. (2016). Cell cycle S phase markers are expressed in cerebral neuron nuclei of cats infected by the Feline Panleukopenia Virus. Cell cycle (Georgetown, Tex.), 15(24), 3482-3489. doi:10.1080/15384101.2016.1249546
  7. 32. Audouard, E., Houben, S., Masaracchia, C., Yilmaz, Z., Suain, V., Authelet, M., De Decker, R., Buée, L., Boom, A., Leroy, K., Ando, K., & Brion, J. P. (2016). High-Molecular-Weight Paired Helical Filaments from Alzheimer Brain Induces Seeding of Wild-Type Mouse Tau into an Argyrophilic 4R Tau Pathology in Vivo. The American journal of pathology, 186(10), 2709-2722. doi:10.1016/j.ajpath.2016.06.008
  8. 33. Ando, K., Tomimura, K., Sazdovitch, V., Suain, V., Yilmaz, Z., Authelet, M., Ndjim, M., Vergara Panos, C., Belkouch, M., Potier, M.-C., Duyckaerts, C., & Brion, J. P. (2016). Level of PICALM, a key component of clathrin-mediated endocytosis, is correlated with levels of phosphotau and autophagy-related proteins and is associated with tau inclusions in AD, PSP and Pick disease. Neurobiology of disease, 94, 32-43. doi:10.1016/j.nbd.2016.05.017
  9. 34. Gilley, J., Ando, K., Seereeram, A., Rodríguez-Martín, T., Pooler, A., Sturdee, L., Anderton, B., Brion, J. P., Hanger, D., & Coleman, M. (2016). Mislocalization of neuronal tau in the absence of tangle pathology in phosphomutant tau knockin mice. Neurobiology of aging, 39, 1-18. doi:10.1016/j.neurobiolaging.2015.11.028
  10. 35. Gilissen, E., Leroy, K., Yilmaz, Z., Kövari, E., Bouras, C., Boom, A., Poncelet, L., Erwin, J. J., Sherwood, C. C., Hof, P., & Brion, J. P. (2016). A neuronal aging pattern unique to humans and common chimpanzees. Brain Structure and Function (Print Edition), 221(1), 647-664. doi:10.1007/s00429-014-0931-5
  11. 36. Bondulich, M. M., Guo, T., Meehan, C., Manion, J., Rodríguez-Martín, T., Mitchell, J. C., Hortobagyi, T., Yankova, N., Stygelbout, V., Brion, J. P., Noble, W., & Hanger, D. (2016). Tauopathy induced by low level expression of a human brain-derived tau fragment in mice is rescued by phenylbutyrate. Brain, 139(Pt 8), 2290-2306. doi:10.1093/brain/aww137
  12. 37. Audouard, E., Van Hees, L., Suain, V., Yilmaz, Z., Poncelet, L., Leroy, K., & Brion, J. P. (2015). Motor Deficit in a Tauopathy Model Is Induced by Disturbances of Axonal Transport Leading to Dying-Back Degeneration and Denervation of Neuromuscular Junctions. The American journal of pathology, 185(10), 2685-2697. doi:10.1016/j.ajpath.2015.06.011

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