Articles dans des revues avec comité de lecture (74)
25.
Machelart, A., Khadrawi, A., Demars, A., Willemart, K., De Trez, C., Letesson, J.-J., & Muraille, E. (2017). Chronic Brucella infection induces selective and persistent interferon gammadependent alterations of marginal zone macrophages in the spleen. Infection and immunity, 85(11), e00115-17. doi:10.1128/IAI.00115-1726.
Cnops, J., Kauffmann, F., De Trez, C., Baltz, T., Keirsse, J., Radwanska, M., Muraille, E., & Magez, S. (2016). Maintenance of B cells during chronic murine Trypanosoma brucei gambiense infection. Parasite immunology, 38(10), 642-647. doi:10.1111/pim.1234427.
Kauffmann, F., Dumetz, F., Hendrickx, S., Muraille, E., Dujardin, J.-C., Maes, L., Magez, S., & De Trez, C. (2016). In vivo characterization of two additional Leishmania donovani strains using the murine and hamster model. Parasite immunology, 38(5), 290-302. doi:10.1111/pim.1231628.
Hanot Mambres, D., Machelart, A., Potemberg, G., De Trez, C., Ryffel, B., Letesson, J.-J., & Muraille, E. (2016). Identification of immune effectors essential to the control of primary and secondary intranasal infection with brucella melitensis in mice. The Journal of immunology, 196(9), 3780-3793. doi:10.4049/jimmunol.150226531.
Hanot Mambres, D., Machelart, A., Vanderwinden, J.-M., Muraille, E., De Trez, C., Ryffel, B., & Letesson, J.-J. (2015). In Situ Characterization of Splenic Brucella melitensis Reservoir Cells during the Chronic Phase of Infection in Susceptible Mice. PloS one, 10(9), e0137835. doi:10.1371/journal.pone.013783532.
Vitry, M.-A., Hanot Mambres, D., Deghelt, M., Hack, K., Machelart, A., Lhommé, F., Vanderwinden, J.-M., Vermeersch, M., De Trez, C., Perez-Morga, D., Letesson, J.-J., & Muraille, E. (2014). Brucella melitensis invades murine erythrocytes during infection. Infection and immunity, 82(9), 3927-3938. doi:10.1128/IAI.01779-1433.
Vitry, M.-A., Hanot Mambres, D., De Trez, C., Akira, S., Ryffel, B., Letesson, J.-J., & Muraille, E. (2014). Humoral Immunity and CD4+ Th1 Cells Are Both Necessary for a Fully Protective Immune Response upon Secondary Infection with Brucella melitensis. The Journal of immunology, 192(8), 3740-3752. doi:10.4049/jimmunol.1302561