Articles dans des revues avec comité de lecture (25)
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Kim, S. Y., Lee, J.-H., Merrins, M. M., Gavrilova, O., Bisteau, X., Kaldis, P., Satin, L. L., & Rane, S. S. (2017). Loss of Cyclin-dependent Kinase 2 in the Pancreas Links Primary β-Cell Dysfunction to Progressive Depletion of β-Cell Mass and Diabetes. The Journal of biological chemistry, 292(9), 3841-3853. doi:10.1074/jbc.M116.75407714.
Jayapal, S. R., Ang, H. Y.-K., Wang, C. Q., Bisteau, X., Caldez, M. M., Xuan, G. X., Yu, W., Tergaonkar, V., Osato, M., Lim, B., & Kaldis, P. (2016). Cyclin A2 regulates erythrocyte morphology and numbers. Cell cycle (Georgetown, Tex.), 15(22), 3070-3081. doi:10.1080/15384101.2016.123454615.
Diril, K. M., Bisteau, X., Kitagawa, M., Caldez, M. M., Wee, S., Gunaratne, J., Lee, S. H., & Kaldis, P. (2016). Loss of the Greatwall Kinase Weakens the Spindle Assembly Checkpoint. PLOS genetics, 12(9), e1006310. doi:10.1371/journal.pgen.100631016.
Chauhan, S., Diril, K. M., Lee, J. H. S., Bisteau, X., Manoharan, V., Adhikari, D., Ratnacaram, C. K., Janela, B., Noffke, J., Ginhoux, F., Coppola, V., Liu, K., Tessarollo, L., & Kaldis, P. (2016). Cdk2 catalytic activity is essential for meiotic cell division in vivo. Biochemical journal, 473(18), 2783-2798. doi:10.1042/BCJ2016060717.
Heijink, A. M., Blomen, V. V., Bisteau, X., Degener, F., Matsushita, F. Y., Kaldis, P., Foijer, F., & van Vugt, M. A. T. M. M. (2015). A haploid genetic screen identifies the G1/S regulatory machinery as a determinant of Wee1 inhibitor sensitivity. Proceedings of the National Academy of Sciences of the United States of America, 112(49), 15160-15165. doi:10.1073/pnas.150528311218.
Jayapal, S. R., Wang, C. Q., Bisteau, X., Caldez, M. M., Lim, S., Tergaonkar, V., Osato, M., & Kaldis, P. (2015). Hematopoiesis specific loss of Cdk2 and Cdk4 results in increased erythrocyte size and delayed platelet recovery following stress. Haematologica, 100(4), 431-438. doi:10.3324/haematol.2014.10646822.
Bisteau, X., Paternot, S., Colleoni, B., Ecker, K., Coulonval, K., De Groote, P., Declercq, W., Hengst, L., & Roger, P. P. (2013). CDK4 T172 phosphorylation is central in a CDK7-dependent bidirectional CDK4/CDK2 interplay mediated by p21 phosphorylation at the restriction point. PLOS genetics, 9(5), e1003546. doi:10.1371/journal.pgen.100354623.
Farhang Ghahremani, M., Goossens, S., Nittner, D., Bisteau, X., Bartunkova, S., Zwolinska, A., Hulpiau, P., Haigh, K., Haenebalcke, L., Drogat, B., Jochemsen, A., Roger, P. P., Marine, J.-C., & Haigh, J. J. (2013). p53 promotes VEGF expression and angiogenesis in the absence of an intact p21-Rb pathway. Cell death and differentiation. doi:10.1038/cdd.2013.1224.
Paternot, S., Bockstaele, L., Bisteau, X., Kooken, H., Coulonval, K., & Roger, P. P. (2010). Rb inactivation in cell cycle and cancer: the puzzle of highly regulated activating phosphorylation of CDK4 versus constitutively active CDK-activating kinase. Cell cycle (Georgetown, Tex.), 9(4), 689-699.