Articles dans des revues avec comité de lecture (56)

  1. 13. Ando, K., De Decker, R., Vergara Panos, C., Yilmaz, Z., Mansour, S., Suain, V., Sleegers, K., De Fisenne, M.-A., Houben, S., Potier, M.-C., Duyckaerts, C., Watanabe, T., Buée, L., Leroy, K., & Brion, J. P. (2020). Picalm reduction exacerbates tau pathology in a murine tauopathy model. Acta Neuropathologica, 139(4), 773-789. doi:10.1007/s00401-020-02125-x
  2. 14. Ando, K., Houben, S., Homa, M., De Fisenne, M.-A., Potier, M.-C., Erneux, C., Brion, J. P., & Leroy, K. (2020). Alzheimer's Disease: Tau Pathology and Dysfunction of Endocytosis. Frontiers in Molecular Neuroscience, 13, 583755. doi:10.3389/fnmol.2020.583755
  3. 15. Houben, S., Leroy, K., Ando, K., Yilmaz, Z., Widomski, C., Buée, L., & Brion, J. P. (2019). Genetic ablation of tau in postnatal neurons rescues decreased adult hippocampal neurogenesis in a tauopathy model. Neurobiology of disease, 127, 131-141. doi:10.1016/j.nbd.2019.02.021
  4. 16. Poncelet, L., Ando, K., Vergara Panos, C., Mansour, S., Suain, V., Yilmaz, Z., Reygel, A., Gilissen, E., Brion, J. P., & Leroy, K. (2019). A 4R tauopathy develops without amyloid deposits in aged cat brains. Neurobiology of aging, 81, NBA10594, 200-212. doi:10.1016/j.neurobiolaging.2019.05.024
  5. 17. Pierrot, N., Ris, L., Stancu, I.-C., Doshina, A., Ribeiro, F., Tyteca, D., Baugé, E., Lalloyer, F., Malong, L., Schakman, O., Leroy, K., Kienlen-Campard, P. K., Gailly, P., Brion, J. P., Dewachter, I., Staels, B., & Octave, J. N. (2019). Sex-regulated gene dosage effect of PPARα on synaptic plasticity. Life science alliance, 2(2). doi:10.26508/lsa.201800262
  6. 18. Vergara Panos, C., Houben, S., Suain, V., Yilmaz, Z., De Decker, R., Vanden Dries, V., Boom, A., Mansour, S., Leroy, K., Ando, K., & Brion, J. P. (2019). Amyloid-β pathology enhances pathological fibrillary tau seeding induced by Alzheimer PHF in vivo. Acta Neuropathologica, 137(3), 397-412. doi:10.1007/s00401-018-1953-5
  7. 19. Vanden Dries, V., Stygelbout, V., Pierrot, N., Yilmaz, Z., Suain, V., De Decker, R., Buée, L., Octave, J. N., Brion, J. P., & Leroy, K. (2017). Amyloid precursor protein reduction enhances the formation of neurofibrillary tangles in a mutant tau transgenic mouse model. Neurobiology of aging. doi:10.1016/j.neurobiolaging.2017.03.031
  8. 20. Espuny Camacho, I. M., Arranz, A. M., Fiers, M., Snellinx, A., Ando, K., Munck, S., Bonnefont, J., Lambot, L., Corthout, N., Omodho, L., Vanden Eynden, E., Radaelli, E., Tesseur, I., Wray, S., Ebneth, A., Hardy, J., Leroy, K., Brion, J. P., Vanderhaeghen, P., & De Strooper, B. (2017). Hallmarks of Alzheimer's Disease in Stem-Cell-Derived Human Neurons Transplanted into Mouse Brain. Neuron, 93(5), 1066-1081.e8. doi:10.1016/j.neuron.2017.02.001
  9. 21. Audouard, E., Houben, S., Masaracchia, C., Yilmaz, Z., Suain, V., Authelet, M., De Decker, R., Buée, L., Boom, A., Leroy, K., Ando, K., & Brion, J. P. (2016). High-Molecular-Weight Paired Helical Filaments from Alzheimer Brain Induces Seeding of Wild-Type Mouse Tau into an Argyrophilic 4R Tau Pathology in Vivo. The American journal of pathology, 186(10), 2709-2722. doi:10.1016/j.ajpath.2016.06.008
  10. 22. Gilissen, E., Leroy, K., Yilmaz, Z., Kövari, E., Bouras, C., Boom, A., Poncelet, L., Erwin, J. J., Sherwood, C. C., Hof, P., & Brion, J. P. (2016). A neuronal aging pattern unique to humans and common chimpanzees. Brain Structure and Function (Print Edition), 221(1), 647-664. doi:10.1007/s00429-014-0931-5
  11. 23. Audouard, E., Van Hees, L., Suain, V., Yilmaz, Z., Poncelet, L., Leroy, K., & Brion, J. P. (2015). Motor Deficit in a Tauopathy Model Is Induced by Disturbances of Axonal Transport Leading to Dying-Back Degeneration and Denervation of Neuromuscular Junctions. The American journal of pathology, 185(10), 2685-2697. doi:10.1016/j.ajpath.2015.06.011
  12. 24. Frédérick, C., Ando, K., Leroy, K., Heraud, C., Suain, V., Buée, L., & Brion, J. P. (2014). Rapamycin Ester Analog CCI-779/Temsirolimus Alleviates Tau Pathology and Improves Motor Deficit in Mutant Tau Transgenic Mice. Journal of Alzheimer's disease. doi:10.3233/JAD-142097

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