Articles dans des revues avec comité de lecture (56)

  1. 1. Meyerovich, K., Ortis, F., & Cardozo, A. K. (2018). The non-canonical NF-κB pathway and its contribution to β-cell failure in diabetes. Journal of molecular endocrinology, 61(2), F1-F6. doi:10.1530/JME-16-0183
  2. 2. Nardelli, T. R., Vanzela, E. C., Benedicto, K. K., Brozzi, F., Fujita, A., Cardozo, A. K., Eizirik, D. L., Boschero, A. C., & Ortis, F. (2018). Prolactin protects against cytokine-induced beta-cell death by NFκB and JNK inhibition. Journal of molecular endocrinology, 61(1), 25-36. doi:10.1530/JME-16-0257
  3. 3. Meyerovich, K., Ortis, F., & Cardozo, A. K. (2018). The non-canonical NF-κB pathway and its contribution to β-cell failure in diabetes. Journal of molecular endocrinology. doi:10.1530/JME-16-0183
  4. 4. Lambelet, M., Terra, L. F., Fukaya, M., Meyerovich, K., Labriola, L. L., Cardozo, A. K., & Allagnat, F. (2018). Dysfunctional autophagy following exposure to pro-inflammatory cytokines contributes to pancreatic β-cell apoptosis. Cell death and disease, 9(2), 96. doi:10.1038/s41419-017-0121-5
  5. 5. Papin, J., Zummo, F. P., Pachera, N., Guay, C., Regazzi, R., Cardozo, A. K., & Herchuelz, A. (2018). Na+/Ca2+ Exchanger a Druggable Target to Promote β-Cell Proliferation and Function. Journal of the Endocrine Society, 2(7), 631–645. doi:10.1210/js.2017-00370
  6. 6. Papin, J., Zummo, F. P., Pachera, N., Guay, C., Regazzi, R., Cardozo, A. K., & Herchuelz, A. (2018). Na+/Ca2+ exchanger a druggable target to promote β-cellproliferation and function. Journal of the Endocrine Society, 2(7), 631-645. doi:10.1210/JS.2017-00370
  7. 7. Meyerovich, K., Moretti Violato, N., Fukaya, M., Dirix, V., Pachera, N., Marselli, L., Marchetti, P., Strasser, A., Eizirik, D., & Cardozo, A. K. (2017). MCL-1 is a Key Anti-Apoptotic Protein in Human and Rodent Pancreatic Beta cells. Diabetes (New York, N.Y.), 66(9), 2446-2458. doi:10.2337/db16-1252
  8. 8. Meyerovich, K., Ortis, F., Allagnat, F., & Cardozo, A. K. (2016). Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. Journal of molecular endocrinology, 57(1), R1-R17. doi:10.1530/JME-15-0306
  9. 9. Meyerovich, K., Fukaya, M., Terra, L. L., Ortis, F., Eizirik, D. L., & Cardozo, A. K. (2016). The non-canonical NF-kB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro. Diabetologia, 59(3), 512-521. doi:10.1007/s00125-015-3817-z
  10. 10. Fukaya, M., Brorsson, C., Meyerovich, K., Catrysse, L., Delaroche, D., Vanzela, E. C., Ortis, F., Beyaert, R., Nielsen, L. B., Andersen, M. L. M. M., Mortensen, H. B. I. H., Pociot, F. M., Van Loo, G., Størling, J., & Cardozo, A. K. (2016). A20 inhibits β-cell apoptosis by multiple mechanisms and predicts residual β-cell function in type 1 diabetes. Molecular endocrinology, 30(1), 48-61. doi:10.1210/me.2015-1176
  11. 11. Pachera, N., Papin, J., Zummo, F. P., Rahier, J., Mast, J., Meyerovich, K., Cardozo, A. K., & Herchuelz, A. (2015). Heterozygous inactivation of plasma membrane Ca2+-ATPase in mice increases glucose-induced insulin release and beta cell proliferation, mass and viability. Diabetologia, 58(12), 2843-2850. doi:10.1007/s00125-015-3745-y
  12. 12. Catrysse, L., Fukaya, M., Sze, M., Meyerovich, K., Beyaert, R., Cardozo, A. K., & Van Loo, G. (2015). A20 deficiency sensitizes pancreatic beta cells to cytokine-induced apoptosis in vitro but does not influence type 1 diabetes development in vivo. Cell death & disease, 6, e1918. doi:10.1038/cddis.2015.301

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