Articles dans des revues avec comité de lecture (21)
1.
Hubesch, G., Dewachter, C., Chomette, L., Hupkens, E., Jespers, P., Vegh, G., Doppler, M., Sheikh Mohammad, U., Thiriard, A., Remmelink, M., Vachiery, J.-L., McEntee, K., & Dewachter, L. (2024). Early Alteration of Right Ventricle-Pulmonary Artery Coupling in Experimental Heart Failure With Preserved Ejection Fraction. Journal of the American Heart Association, 13(11), e032201. doi:10.1161/JAHA.123.0322012.
El Oumeiri, B. G., Dewachter, L., Van De Borne, P., Hubesch, G., Melot, C., Jespers, P., Stefanidis, C., McEntee, K., & Vanden Eynden, F. (2023). Altered Left Ventricular Rat Gene Expression Induced by the Myosin Activator Omecamtiv Mecarbil. Genes, 14(1). doi:10.3390/genes140101223.
El Oumeiri, B. G., Van De Borne, P., Hubesch, G., Jespers, P., Dewachter, L., Stefanidis, C., McEntee, K., & Vanden Eynden, F. (2022). Detection of soluble suppression of tumorigenicity 2 and N-terminal B-type natriuretic peptide in a rat model of aortic regurgitation: differential responses to omecamtiv mecarbil. Journal of basic & clinical physiology & pharmacology, 33(6), 743-750. doi:10.1515/jbcpp-2022-02154.
Hubesch, G., Hanthazi, A., Acheampong, A., Chomette, L., Lasolle, H., Hupkens, E., Jespers, P., Vegh, G., Watu Malu Wembonyama, C., Verhoeven, C., Dewachter, C., Vachiery, J.-L., McEntee, K., & Dewachter, L. (2022). A Preclinical Rat Model of Heart Failure With Preserved Ejection Fraction With Multiple Comorbidities. Frontiers in Cardiovascular Medicine, 8. doi:10.3389/fcvm.2021.8098855.
Boudart, C., Su, F., Pitisci, L., Dhoine, A., Duranteau, O., Jespers, P., Herpain, A., Vanderpool, R., Brimioulle, S., Creteur, J., Naeije, R., Van Obbergh, L., & Dewachter, L. (2021). Early Hyperdynamic Sepsis Alters Coronary Blood Flow Regulation in Porcine Fecal Peritonitis. Frontiers in physiology, 12, 754570. doi:10.3389/fphys.2021.7545706.
El Oumeiri, B. G., Van De Borne, P., Hubesch, G., Herpain, A., Annoni, F., Jespers, P., Stefanidis, C., Mc Entee, K., & Vanden Eynden, F. (2021). The myosin activator omecamtiv mecarbil improves wall stress in a rat model of chronic aortic regurgitation. Physiological Reports, 9(16), e14988. doi:10.14814/phy2.149887.
Hanthazi, A., Jespers, P., Vegh, G., Dubois, C., Hubesch, G., Springael, J.-Y., Dewachter, L., & McEntee, K. (2020). Chemerin Added to Endothelin-1 Promotes Rat Pulmonary Artery Smooth Muscle Cell Proliferation and Migration. Frontiers in physiology, 11, 926. doi:10.3389/fphys.2020.009268.
Hanthazi, A., Jespers, P., Vegh, G., Degroot, G.-N., Springael, J.-Y., Lybaert, P., Dewachter, L., & McEntee, K. (2019). Chemerin influences endothelin- and serotonin-induced pulmonary artery vasoconstriction in rats. Life sciences, 231, 116580. doi:10.1016/j.lfs.2019.1165809.
Oumeiri, B. E., McEntee, K., Annoni, F., Herpain, A., Vanden Eynden, F., Jespers, P., Van Nooten, G., & Van De Borne, P. (2018). Effects of the cardiac myosin activator Omecamtiv-mecarbil on severe chronic aortic regurgitation in Wistar rats. BMC cardiovascular disorders, 18(1), 99. doi:10.1186/s12872-018-0831-310.
Gomart, S., Gaudreau Ménard, C., Jespers, P., Dilek, G., Hupkens, E., Hanthazi, A., Naeije, R., Melot, C., Labranche, N., Dewachter, L., & McEntee, K. (2017). Leptin-Induced Endothelium-Independent Vasoconstriction in Thoracic Aorta and Pulmonary Artery of Spontaneously Hypertensive Rats: Role of Calcium Channels and Stores. PloS one, 12(1), e0169205. doi:10.1371/journal.pone.016920511.
Damoiseaux, C., Merveille, A.-C., Krafft, E., Mendes Da Costa, A., Gomart, S., Jespers, P., Michaux, C., Clercx, C., Verhoeven, C., & McEntee, K. (2014). Effect of physiological determinants and cardiac disease on plasma adiponectin concentration in dogs. Journal of veterinary internal medicine, 28, 1738-1745. doi:10.1111/jvim.1243312.
Gomart, S., Damoiseaux, C., Jespers, P., Labranche, N., Pochet, S., Michaux, C., Berkenboom, G., Naeije, R., McEntee, K., & Dewachter, L. (2014). Pulmonary vasoreactivity in spontaneously hypertensive rats : effects of endothelin-1 and leptin. Respiratory research, 15(1), 12. doi:10.1186/1465-9921-15-12