Articles dans des revues avec comité de lecture (58)
  1. 1. Bastide, L., Imbault, V., Perrotta, G., Borrelli, S., Elands, S., Van Pesch, V., Borràs, E., Sabido, E., Gaspard, N., Communi, D., & Bisteau, X. (2026). Proteomic Signatures of Conversion Risk and Disease Severity in Multiple Sclerosis. medRxiv : the preprint server for health sciences. doi:10.64898/2026.03.25.26348613
  2. 2. Dionisi, C., Chazalon, M., Rai, M., Keime, C., Imbault, V., Communi, D., Puccio, H., Schiffmann, S. N., & Pandolfo, M. (2023). Proprioceptors-enriched neuronal cultures from induced pluripotent stem cells from Friedreich ataxia patients show altered transcriptomic and proteomic profiles, abnormal neurite extension, and impaired electrophysiological properties. Brain Communications, 5(1), fcad007. doi:10.1093/braincomms/fcad007
  3. 3. Santacruz, C. A., Vincent, J. L., Imbault, V., Bruneau, M., Creteur, J., Brimioulle, S., Vincent, R., Communi, D., & Taccone, F. S. (2023). Cerebral apolipoprotein E and amyloid precursor-like protein 1 as risk factors for chronic neurodegeneration after non-traumatic acute brain injury (ABI). Critical care, 27(1), 249. doi:10.1186/s13054-023-04538-y
  4. 4. Santacruz, C. A., Vincent, J. L., Duitama, J., Bautista, E., Imbault, V., Bruneau, M., Creteur, J., Brimioulle, S., Communi, D., & Taccone, F. S. (2023). vCSF Danger-associated Molecular Patterns After Traumatic and Nontraumatic Acute Brain Injury: A Prospective Study. Journal of neurosurgical anesthesiology. doi:10.1097/ANA.0000000000000916
  5. 5. Sidiras, P., Lechanteur, J., Imbault, V., Sokolova, T., Durez, P., Gangji, V., Communi, D., & Rasschaert, J. (2022). Human carbamylome description identifies carbamylated α2-macroglobulin and hemopexin as two novel autoantigens in early rheumatoid arthritis. Rheumatology (Online), 61(7), 2826-2834. doi:10.1093/rheumatology/keab838
  6. 6. Imbault, V., Dionisi, C., Naeije, G., Communi, D., & Pandolfo, M. (2022). Cerebrospinal Fluid Proteomics in Friedreich Ataxia Reveals Markers of Neurodegeneration and Neuroinflammation. Frontiers in Neuroscience, 16, 885313. doi:10.3389/fnins.2022.885313
  7. 7. Santacruz, C., Vincent, J. L., Duitama, J., Bautista, E., Imbault, V., Bruneau, M., Creteur, J., Brimioulle, S., Communi, D., & Taccone, F. S. (2022). The Cerebrospinal Fluid Proteomic Response to Traumatic and Nontraumatic Acute Brain Injury: A Prospective Study. Neurocritical care. doi:10.1007/s12028-022-01507-1
  8. 8. Sidiras, P., Lechanteur, J., Imbault, V., Sokolova, T., Durez, P., Gangji, V., Communi, D., & Rasschaert, J. (2021). Human carbamylome description identifies carbamylated α2-macroglobulin and hemopexin as two novel autoantigens in early rheumatoid arthritis. Rheumatology. doi:10.1093/rheumatology/keab838
  9. 9. Santacruz, C. A., Vincent, J. L., Bader, A., Rincón-Gutiérrez, L. L., Dominguez-Curell, C., Communi, D., & Taccone, F. S. (2021). Association of cerebrospinal fluid protein biomarkers with outcomes in patients with traumatic and non-traumatic acute brain injury: systematic review of the literature. Critical care, 25(1), 278. doi:10.1186/s13054-021-03698-z
  10. 10. Sidiras, P., Spruyt, D., Gangji, V., Imbault, V., Sokolova, T., Durez, P., Communi, D., Rasschaert, J., & Badot, V. (2020). Antibodies against carbamylated proteins: prevalence and associated disease characteristics in Belgian patients with rheumatoid arthritis or other rheumatic diseases. Scandinavian journal of rheumatology, 1-6. doi:10.1080/03009742.2020.1798500
  11. 11. Karadurmus, D., Rial, D., De Backer, J.-F., Communi, D., de Kerchove d'Exaerde, A., & Schiffmann, S. N. (2019). GPRIN3 Controls Neuronal Excitability, Morphology, and Striatal-Dependent Behaviors in the Indirect Pathway of the Striatum. The Journal of neuroscience, 39(38), 7513-7528. doi:10.1523/JNEUROSCI.2454-18.2019
  12. 12. Vande Catsyne, C. A., Sayyed, S. A., Molina-Ortiz, P., Moes, B., Communi, D., Muller, J., Heusschen, R., Caers, J., Azzi, A., Erneux, C., & Schurmans, S. (2019). Altered chondrocyte differentiation, matrix mineralization and MEK-Erk1/2 signaling in an INPPL1 catalytic knock-out mouse model of opsismodysplasia. Advances in biological regulation., 100651. doi:10.1016/j.jbior.2019.100651
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