• Citer

Tumor necrosis factor alpha decreases, and interleukin-10 increases, the sensitivity of human monocytes to dexamethasone: potential regulation of the glucocorticoid receptor.

par Franchimont, Denis ;Martens, H.;Hagelstein, M T;Louis, Edouard;Dewe, W;Chrousos, G P;Belaiche, Jacques;Geenen, Vincent
Référence The Journal of clinical endocrinology and metabolism, 84, 8, page (2834-2839)
Publication Publié, 1999-08

Article révisé par les pairs

• Détails
• Contenu

Titre:	Tumor necrosis factor alpha decreases, and interleukin-10 increases, the sensitivity of human monocytes to dexamethasone: potential regulation of the glucocorticoid receptor.
Auteur:	Franchimont, Denis; Martens, H.; Hagelstein, M T; Louis, Edouard; Dewe, W; Chrousos, G P; Belaiche, Jacques; Geenen, Vincent
Informations sur la publication:	The Journal of clinical endocrinology and metabolism, 84, 8, page (2834-2839)
Statut de publication:	Publié, 1999-08
Sujet CREF:	Sciences bio-médicales et agricoles
MeSH keywords:	Adult
	Cell Line
	Dexamethasone -- pharmacology
	Humans
	Interleukin 1 Receptor Antagonist Protein
	Interleukin-10 -- biosynthesis
	Interleukin-10 -- pharmacology
	Lipopolysaccharides -- pharmacology
	Male
	Middle Aged
	Monocytes -- chemistry
	Monocytes -- drug effects
	Receptors, Glucocorticoid -- analysis
	Sialoglycoproteins -- biosynthesis
	Tumor Necrosis Factor-alpha -- biosynthesis
	Tumor Necrosis Factor-alpha -- pharmacology
Note générale:	Journal Article
	Research Support, Non-U.S. Gov't
Langue:	Anglais
Identificateurs:	urn:issn:0021-972X
	info:pmid/10443688