par Horckmans, Michael ;Ring, Larisa;Duchene, Johan;Santovito, Donato;Schloss, Maximilian MJ;Drechsler, Maik;Lehners-Weber, Christiane;Soehnlein, Oliver;Steffens, Sabine
Référence European heart journal, 38, 3, page (187-197)
Publication Publié, 2017
Référence European heart journal, 38, 3, page (187-197)
Publication Publié, 2017
Article révisé par les pairs
Résumé : | Acute myocardial infarction (MI) is the leading cause of mortality worldwide. Anti-inflammatory strategies to reduce neutrophil-driven acute post-MI injury have been shown to limit acute cardiac tissue damage. On the other hand, whether neutrophils are required for resolving post-MI inflammation and repair is unknown. |