Titre :
  • BIM Deficiency Protects NOD Mice From Diabetes by Diverting Thymocytes to Regulatory T Cells.
Auteur : Krishnamurthy, Balasubramanian B ; Chee, Jonathan J ; Jhala, Gaurang G ; Trivedi, Prerak P ; Catterall, Tara T ; Selck, Claudia C ; Gurzov, Esteban Nicolas ; Brodnicki, Thomas C TC ; Graham, Kate L KL ; Wali, Jibran A JA ; Zhan, Yifan Y ; Gray, Daniel D ; Strasser, Andreas A ; Allison, Janette J ; Thomas, Helen H.E. ; Kay, Thomas W H TW
Informations sur la publication : Diabetes, 64, 9, (page 3229-3238)
Statut de publication : Publié, 2015-09
Sujet CREF : Sciences bio-médicales et agricoles
Mots-clés MeSH : Animals
Antigens, CD5 -- metabolism
Apoptosis Regulatory Proteins -- genetics -- immunology
Bcl-2-Like Protein 11
Clonal Deletion -- genetics -- immunology
Diabetes Mellitus, Type 1 -- genetics -- immunology
Disease Models, Animal
Glucocorticoid-Induced TNFR-Related Protein -- metabolism
I-kappa B Proteins -- metabolism
Membrane Proteins -- genetics -- immunology
Mice
Mice, Inbred NOD
NF-KappaB Inhibitor alpha
Nuclear Receptor Subfamily 4, Group A, Member 1 -- metabolism
Phosphorylation
Proto-Oncogene Proteins -- genetics -- immunology
T-Lymphocytes, Regulatory -- immunology
Thymocytes -- immunology -- metabolism
Langue :
  • Anglais
Identificateurs : urn:issn:0012-1797 
info:doi/10.2337/db14-1851
info:pii/db14-1851
info:pmid/25948683