Titre:
  • BIM Deficiency Protects NOD Mice From Diabetes by Diverting Thymocytes to Regulatory T Cells.
Auteur:Krishnamurthy, Balasubramanian; Chee, Jonathan; Jhala, Gaurang; Trivedi, Prerak; Catterall, Tara; Selck, Claudia; Gurzov, Esteban Nicolas; Brodnicki, Thomas C; Graham, Kate L; Wali, Jibran A; Zhan, Yifan; Gray, Daniel; Strasser, Andreas; Allison, Janette; Thomas, Helen H.E.; Kay, Thomas W H TW
Informations sur la publication:Diabetes, 64, 9, page (3229-3238)
Statut de publication:Publié, 2015-09
Sujet CREF:Sciences bio-médicales et agricoles
MeSH keywords:Animals
Antigens, CD5 -- metabolism
Apoptosis Regulatory Proteins -- genetics -- immunology
Bcl-2-Like Protein 11
Clonal Deletion -- genetics -- immunology
Diabetes Mellitus, Type 1 -- genetics -- immunology
Disease Models, Animal
Glucocorticoid-Induced TNFR-Related Protein -- metabolism
I-kappa B Proteins -- metabolism
Membrane Proteins -- genetics -- immunology
Mice
Mice, Inbred NOD
NF-KappaB Inhibitor alpha
Nuclear Receptor Subfamily 4, Group A, Member 1 -- metabolism
Phosphorylation
Proto-Oncogene Proteins -- genetics -- immunology
T-Lymphocytes, Regulatory -- immunology
Thymocytes -- immunology -- metabolism
Langue:Anglais
Identificateurs:urn:issn:0012-1797
info:doi/10.2337/db14-1851
info:pii/db14-1851
info:pmid/25948683