par Lichtenstein, Laeticia;Perret, Bertrand;Martinez, Laurent O;Serhan, Nizar;Espinosa-Delgado, Sara;Fabre, Aurélie C;Annema, Wijtske;Tietge, Uwe J F;Robaye, Bernard ;Boeynaems, Jean-Marie ;Laffargue, Muriel
Référence Cardiovascular Research, 106, 2, page (314-323)
Publication Publié, 2015-05
Référence Cardiovascular Research, 106, 2, page (314-323)
Publication Publié, 2015-05
Article révisé par les pairs
Résumé : | Aims High-density lipoproteins (HDLs) protect against atherosclerosis mainly due to their function in hepatobiliary reverse cholesterol transport (RCT). This is a process whereby excess cholesterol from peripheral tissues is transported by HDL particles to the liver for further metabolism and biliary excretion. Hepatic uptake of HDL holoparticles involves the P2Y |