par Delince, Philippe 
;Krallis, Panagiotis ;Descamps, Pierre-Yves ;Fabeck, Laurent ;Hardy, Dominique
Référence Arthroscopy, 14, 8, page (869-876)
Publication Publié, 1998
;Krallis, Panagiotis ;Descamps, Pierre-Yves ;Fabeck, Laurent ;Hardy, Dominique Référence Arthroscopy, 14, 8, page (869-876)
Publication Publié, 1998
Article révisé par les pairs
| Résumé : | After anterior cruciate ligament (ACL) reconstruction using a patellar- tendon autograft, 65 patients underwent second-look arthroscopy in conjunction with hardware removal. In 23 patients, hypertrophic tissue was found in the anterior part of the knee. This tissue presented different aspects, from a well-synovialized nodule to a more disorganized fibrous tissue according to patients' complaints. Endoscopic resection of this offending tissue was generally sufficient to obtain a satisfactory result. In patients presenting a loss of extension, the notch frequently had to be enlarged. We have found a multifactorial pathogenesis to be likely: the nodule is a natural fibroproliferative tissue process originating either from drilling debris from the tibial tunnel or from remnants of the ACL stump and, more rarely, from broken graft fibers. Sometimes, when the graft is malpositioned, the scar tissue can result from repeated graft impingement on the notch at terminal extension. Formation of this aberrant tissue should be prevented by proper positioning of the graft, by enlargement of the narrowed notch in chronic cases, by using drills of increasing diameters to avoid production of osteocartilaginous fragments, by meticulous resection of all drilling debris and ACL remnants around the tibial tunnel, and by enlarging the notch roof if any contact with the graft is present when the knee is fully extended intraoperatively. |
